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Pharmacology - Glucocorticoids
 
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http://armandoh.org/ https://www.facebook.com/ArmandoHasudungan Support me: http://www.patreon.com/armando Instagram: http://instagram.com/armandohasudungan Twitter: https://twitter.com/Armando71021105 Aracidonic Acid: http://www.youtube.com/watch?v=VSc9U8JaWYg Osteoporosis: http://www.youtube.com/watch?v=2cz2rkBy2rg
Views: 231515 Armando Hasudungan
Corticosteroid Mnemonics in Pharmacology for Medical  and Dental Professional Exams ,Session 1
 
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This video deals with Quick n easy way to memorise about Corticosteroid drugs.Corticosteroids are a class of steroid hormones that are produced in the adrenal cortex of vertebrates, as well as the synthetic analogues of these hormones. Two main classes of corticosteroids, glucocorticoidsand mineralocorticoids, are involved in a wide range of physiologic processes, including stress response, immune response, and regulation of inflammation, carbohydratemetabolism, protein catabolism, blood electrolyte levels, and behavior.[1]Synthetic pharmaceutical drugs with corticosteroid-like effects are used in a variety of conditions, ranging from brain tumors to skin diseases. Dexamethasone and its derivatives are almost pure glucocorticoids, while prednisone and its derivatives have some mineralocorticoid action in addition to the glucocorticoid effect. Fludrocortisone(Florinef) is a synthetic mineralocorticoid. Hydrocortisone (cortisol) is typically used for replacement therapy, e.g. for adrenal insufficiency and congenital adrenal hyperplasia. Medical conditions treated with systemic corticosteroids:[3][4] Allergy and respirology medicineAsthma (severe exacerbations)Chronic obstructive pulmonary disease(COPD)Allergic rhinitisAtopic dermatitisHivesAngioedemaAnaphylaxisFood allergiesDrug allergiesNasal polypsHypersensitivity pneumonitisSarcoidosisEosinophilic pneumoniaInterstitial lung diseaseDermatologyPemphigus vulgarisContact dermatitisEndocrinology (usually at physiologic doses)Adrenal insufficiencyCongenital adrenal hyperplasiaGastroenterologyUlcerative colitisCrohn’s diseaseAutoimmune hepatitisHematologyLymphomaLeukemiaHemolytic anemiaIdiopathic thrombocytopenic purpuraMultiple MyelomaRheumatology/ImmunologyRheumatoid arthritisSystemic lupus erythematosusPolymyalgia rheumaticaPolymyositisDermatomyositisPolyarteritisVasculitisOphthalmologyUveitisKeratoconjunctivitisOther conditionsMultiple sclerosisOrgan transplantationNephrotic syndromeChronic hepatitis (flare ups)Cerebral edemaIgG4-related diseaseProstate cancerTendinosisLichen planus Topical formulations are also available for the skin, eyes (uveitis), lungs (asthma), nose (rhinitis), and bowels. Corticosteroids are also used supportively to prevent nausea, often in combination with 5-HT3 antagonists (e.g.ondansetron). Typical undesired effects of glucocorticoids present quite uniformly as drug-induced Cushing's syndrome. Typical mineralocorticoid side-effects are hypertension (abnormally high blood pressure), hypokalemia (low potassium levels in the blood), hypernatremia (high sodium levels in the blood) without causing peripheral edema, metabolic alkalosis and connective tissue weakness.[5] Wound healing or ulcer formation may be ibhibited by the immunosuppressive effects. Clinical and experimental evidence indicates that corticosteroids can cause permanent eye damage by inducing central serous retinopathy (CSR, also known as central serous chorioretinopathy, CSC).[6] A variety of steroid medications, from anti-allergy nasal sprays (Nasonex, Flonase) to topical skin creams, to eye drops (Tobradex), to prednisone have been implicated in the development of CSR.[7][8] Corticosteroids have been widely used in treating people with traumatic brain injury.[9] A systematic review identified 20 randomised controlled trials and included 12,303 participants, then compared patients who received corticosteroids with patients who received no treatment. The authors recommended people with traumatic head injury should not be routinely treated with corticosteroids.[10] Use of corticosteroids has numerous side-effects, some of which may be severe: Neuropsychiatric: steroid psychosis,[13] and anxiety,[14] depression. Therapeutic doses may cause a feeling of artificial well-being ("steroid euphoria").[15] The neuropsychiatric effects are partly mediated by sensitization of the body to the actions of adrenaline. Therapeutically, the bulk of corticosteroid dose is given in the morning to mimic the body's diurnal rhythm; if given at night, the feeling of being energized will interfere with sleep. An extensive review is provided by Flores and Gumina.[16]Cardiovascular: Corticosteroids can cause sodium retention through a direct action on the kidney, in a manner analogous to the mineralocorticoid aldosterone. This can result in fluid retention and hypertension.Metabolic: Corticosteroids cause a movement of body fat to the face and torso, resulting respectively in "moon face" and "buffalo hump". and away from the limbs. Due to the diversion of amino-acids to glucose, they are considered anti-anabolic, and long term therapy can cause muscle wasting[17]Endocrine: By increasing the production of glucose from amino-acid breakdown and opposing the action of insulin, corticosteroids can cause hyperglycemia,[18] insulin resistance and diabetes mellitus.[19]Skeletal: Steroid-induced osteoporosis may be a side-effect of long-term corticosteroid use. Use of inhaled corticosteroids amon
Views: 2016 Dr Anshuman Tripathi
Renal tubular acidosis - causes, symptoms, diagnosis, treatment, pathology
 
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What is renal tubular acidosis (RTA)? RTA is a type of metabolic acidosis caused by the kidneys failure to properly acidify the urine. Find more videos at http://osms.it/more. Hundreds of thousands of current & future clinicians learn by Osmosis. We have unparalleled tools and materials to prepare you to succeed in school, on board exams, and as a future clinician. Sign up for a free trial at http://osms.it/more. Subscribe to our Youtube channel at http://osms.it/subscribe. Get early access to our upcoming video releases, practice questions, giveaways, and more when you follow us on social media: Facebook: http://osms.it/facebook Twitter: http://osms.it/twitter Instagram: http://osms.it/instagram Our Vision: Everyone who cares for someone will learn by Osmosis. Our Mission: To empower the world’s clinicians and caregivers with the best learning experience possible. Learn more here: http://osms.it/mission Medical disclaimer: Knowledge Diffusion Inc (DBA Osmosis) does not provide medical advice. Osmosis and the content available on Osmosis's properties (Osmosis.org, YouTube, and other channels) do not provide a diagnosis or other recommendation for treatment and are not a substitute for the professional judgment of a healthcare professional in diagnosis and treatment of any person or animal. The determination of the need for medical services and the types of healthcare to be provided to a patient are decisions that should be made only by a physician or other licensed health care provider. Always seek the advice of a physician or other qualified healthcare provider with any questions you have regarding a medical condition.
Views: 114732 Osmosis
Shortness of Breath - patients wait 2 years & spend $150K to find real cause (5)
 
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Shortness of breath? Learn more and get a better lung disease treatment plan with top experts. Visit our Online Expert Centers at https://diagnosticdetectives.com/expert-centers/ Exclusive interview / Dr. Anton Titov MD and Professor Dr. Aaron Waxman MD, leading pulmonary arterial hypertension, right heart failure and lung disease expert. Find out exact cause if you have shortness of breath - get a precise diagnosis and best treatment for shortness of breath root cause. - Becoming short of breath is a common symptom, and shortness of breath has many causes. One of those causes could be pulmonary arterial hypertension, a relatively rare situation, but nevertheless a very important disease. How to recognize causes of shortness of breath correctly? So I think that's an important fundamental question because so many people who do complain of shortness of breath are kind of given a diagnosis without a lot of evaluation. And because of that and also because we wanted to actually discover patients with pulmonary hypertension earlier in their disease, we about seven years ago now set up a Dyspnea Evaluation Program. And what we learned very quickly is, #1, that shortness of breath as a complaint is probably second only to pain when people go to their doctors. Whether they go to their primary care, whether they go to a cardiologist or a pulmonologist. And generally those are the three kind of doctors that people with shortness of breath go to. We then learned very quickly that if you're young - and by young I mean less than 55, you'll often be told "Oh, it's asthma". And without any evaluation you'll be given inhalers and sent on your way. If those inhalers don't work - you don't have asthma. But still people were treated [incorrectly, as if they had asthma] If you are older than 55, you were often told you either had COPD [Chronic Obstructive Pulmonary Disease] or probably heart failure. And if you think about it, all three of those diseases are actually very easy to diagnose and rule out, which many of these patients didn't have. So we actually found that when we talk about unexplained dyspnea [shortness of breath], or unexplained exertional intolerance, we only need three tests to put patients into that category. And if they've had pulmonary function testing that is either normal or doesn't explain their limitation, if they've had an echocardiogram that doesn't explain their limitation or is normal and some sort of chest imaging, which whether it's a chest radiograph or a CT scan. Most patients aren't symptomatic at rest. So to expect any of these tests to tell you why someone is short of breath when it's not an obvious problem is asking a lot of the tests. So we actually developed a program here based on invasive cardiopulmonary exercise testing, where we put in a right heart catheter and we specifically use a Paceport Swan-Ganz catheter, so that we can measure pressures out of the right atrium, right ventricle, and pulmonary artery, we wedge that catheter every minute during the test. And we can get a left atrial pressure. And then we also put in a radial artery catheter to measure pressure. And we can then sample blood from both of these catheters. And then we do a full cardiopulmonary exercise test. And it's an incremental load test, so we start out at rest and then we'll do about two minutes of unloaded cycling and then we'll do a ramp protocol depending on the patient's abilities to peak exercise. And then we'll also collect data on the recovery phase, and then usually one hour later we'll get one last set of blood samples. So every minute of this test we're capturing the waveforms off the catheters, we are sampling the catheters, we're measuring gas exchange continuously. So we truly have access to Fick principle physiology.
Pharmacology Tips Loop Diuretics  Lasix
 
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EmpoweRN.com Thank you so much for watching my channel! For the additional resources you can visit here: http://empowern.com/2015/03/pharmacology-loop-diuretics-lasix/ Also, to enter the giveaway all you have to do is: 1. Post a comment 2. Subscribe to the channel. Winner will be announced via email here: http://goo.gl/NO7T7b I would like to thank the video contributors: Rizalyn Joy Gadugdug Maria Salvacion Gonzales Yasmin Hashmi Artem Shestakov And Babar Hayatrana Disclaimer: These videos are intended for entertainment purposes only. Please follow the policy and procedures that your institution requires. Please note that the views, ideas & opinions expressed on this channel and in the videos on this channel are not necessarily of those of my employer or institution. The views expressed on this channel and in the videos channel do not represent medical advice. If you have specific medical concerns, please contact your physician. In order to protect patient privacy, all patient identifiers in all videos have been deleted or altered. The views expressed on this channel and in the videos on this channel are personal opinions. I am not an expert nor do I dispense medical advice or procedural specifications. The information I present is for general knowledge and entertainment purposes only. You need to refer to your own medical director, teachers and protocols for specific treatment information. It is your responsibility to know how best to treat your patient in your jurisdiction. Furosemide (Lasix) - Loop Diuretics Generic Name: Furosemide Brand Name: Lasix Furosemide also known as Lasix is a loop diuretic commonly use to treat fluid retention and edema. Which can be associated with Congestive Heart Failure (CHF), cirrhosis of the liver, and many kidney disorders. It is also sometimes use to treat hypertension alone or in combination with other anti-hypertensive drugs. Diuretics are medications that increases the amount of water that passes through the kidneys as urine, thus, it is often called water pill. Lasix belongs to the group known as Loop diuretics. There a few types of diuretics and loop diuretics are just one type. It works by interfering with the sodium, potassium and chloride symporter. A symporter is a protein membrane that manages the transport of molecules across a cell membrane. The symporter that lasix interacts with is found in the thick ascending limb of the loop of Henle. What it does is………. inhibits the reabsorption of salt (Na+) and Chloride (Cl-). Since the Salt and Chloride will be leaving the body system through the loop of henle this means that water will also follow…. causing more water pass through the kidney. Which will ultimately mean, less fluid remaining in the blood stream. Once Lasix is received the system will then compensate the loss by absorbing any fluid accumulated in the tissues, such as in the lungs or legs, back to the blood stream. This will cause more fluid to be in the blood stream. Which once again pass through the kidney, which will also be excreted in the urine, since the loop diuretic inhibits its reabsorption. This mechanism, is what makes loop diuretics a treatment for fluid retention also known as edema. It can therefore, ease symptoms of edema such as breathlessness caused by congestion of fluid in the lungs which patients diagnosed with CHF, cirrhosis of the liver, nephrotic syndrome and other edematous states may encounter. Lasix is available in forms that can be taken orally and intravenously. When taken intravenously, it is considered to be twice as strong. Parenteral or intravenous Furosemide is indicated when a fast acting and an intense diuresis is needed such as in acute pulmonary edema and cerebral edema. It also indicated when oral therapy is not possible because of problem with absorption in the intestine or for other reasons. Parenteral administration should be observed only in hospital or outpatient clinics. However, in cases of emergency that furosemide should be given right away outside hospital setting, recommended doses should be closely adhered to and patient must be closely monitored. Contraindication: Furosemide should not be taken if your patient cannot urinate. Severe Hypokalemia - is a major contraindication. This means low potassium. Lasix can cause a dramatic decrease in potassium level which could cause lethal cardiac arrhythmias Other contraindications include: Hypotension or low blood pressure Lasix should be used with extreme caution for patients who have a diagnosis of Systemic Lupus Erythematosus because it may May
Views: 85230 EmpoweRN
BETNESOL Tablet Uses Composition Side Effect Precaution How To Uses & Review
 
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BETNESOL Tablet Uses Composition Side Effect Precaution How To Uses & Review Betamethasone is a steroid medication. It is used for a number of diseases including rheumatic disorders such as rheumatoid arthritis and systemic lupus erythematosus, skin diseases such as dermatitis and psoriasis, allergic conditions such as asthma and angioedema, preterm labor to speed the development of the baby's lungs, Crohn's disease, cancers such as leukemia, and along with fludrocortisone for adrenocortical insufficiency, among others. It can be taken by mouth BETNESOL Tablet costs only 9 rs for 20 tablet BETNESOL Tablet is gsk formulation BETNESOL Tablet contains betamethasone side effects include an increased risk of infection, muscle weakness, severe allergic reactions, and psychosis. Long-term use may cause adrenal insufficiency. Stopping the medication suddenly following long-term use may be dangerous Side effects Euphoria Depression Adrenal suppression Hypertension Groupings of fine blood vessels becoming prominent under the skin, petechiae Excessive hair growth (hypertrichosis) Ecchymoses Prolonged use of this medicine on extensive areas of skin, broken or raw skin, skin folds, or underneath airtight dressings may on rare occasions result in enough corticosteroid being absorbed to have side effects on other parts of the body; for example, by causing a decrease in the production of natural hormones by the adrenal glands. Betamethasone is also used prior to delivery of a preterm baby to help prepare the lungs for breathing. However, because betamethasone crosses the placenta, which is required for its beneficial effects, it may also be associated with complications, such as hypoglycemia and leukocytosis in newborns exposed in utero. When injected into the epidural space or the spine, it may cause serious side effects like loss of vision, stroke, and paralysis #onlinemedicine,#medicine,#ayurveda,#ayurved ,#Ayurvedic
Views: 152277 Medicine Reviews
Aldosterone raises blood pressure and lowers potassium | NCLEX-RN | Khan Academy
 
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See how Aldosterone effects the principal cells of the kidney to raise BP and lower potassium. Rishi is a pediatric infectious disease physician and works at Khan Academy. Created by Rishi Desai. Watch the next lesson: https://www.khanacademy.org/test-prep/nclex-rn/rn-renal-system/rn-renal-regulation-of-blood/v/aldosterone-removes-acid-from-the-blood?utm_source=YT&utm_medium=Desc&utm_campaign=Nclex-rn Missed the previous lesson? https://www.khanacademy.org/test-prep/nclex-rn/rn-renal-system/rn-renal-regulation-of-blood/v/angiotensin-2-raises-blood-pressure?utm_source=YT&utm_medium=Desc&utm_campaign=Nclex-rn NCLEX-RN on Khan Academy: A collection of questions from content covered on the NCLEX-RN. These questions are available under a Creative Commons Attribution-NonCommercial-ShareAlike 3.0 United States License (available at http://creativecommons.org/licenses/by-nc-sa/3.0/us/). About Khan Academy: Khan Academy offers practice exercises, instructional videos, and a personalized learning dashboard that empower learners to study at their own pace in and outside of the classroom. We tackle math, science, computer programming, history, art history, economics, and more. Our math missions guide learners from kindergarten to calculus using state-of-the-art, adaptive technology that identifies strengths and learning gaps. We've also partnered with institutions like NASA, The Museum of Modern Art, The California Academy of Sciences, and MIT to offer specialized content. For free. For everyone. Forever. #YouCanLearnAnything Subscribe to Khan Academy’s NCLEX-RN channel: https://www.youtube.com/channel/UCDx5cTeADCvKWgF9x_Qjz3g?sub_confirmation=1 Subscribe to Khan Academy: https://www.youtube.com/subscription_center?add_user=khanacademy
Views: 260847 khanacademymedicine
Hypertension - CRASH! Medical Review Series
 
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(Disclaimer: The medical information contained herein is intended for physician medical licensing exam review purposes only, and are not intended for diagnosis of any illness. If you think you may be suffering from any medical condition, you should consult your physician or seek immediate medical attention.)
Views: 33442 Paul Bolin, M.D.
Fluorine
 
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Fluorine is a chemical element with the symbol F and atomic number 9. It is the lightest halogen and exists as a highly toxic pale yellow diatomic gas at standard conditions. As the most electronegative element, it is extremely reactive: almost all other elements, including some noble gases, form compounds with fluorine. Among the elements, fluorine ranks 24th in universal abundance and 13th in terrestrial abundance. Fluorite, the primary mineral source of fluorine, was first described in 1529; as it was added to metal ores to lower their melting points for smelting, the Latin verb fluo meaning "flow" became associated with it. Proposed as an element in 1810, fluorine proved difficult and dangerous to separate from its compounds, and several early experimenters died or sustained injuries from their attempts. Only in 1886 did French chemist Henri Moissan isolate elemental fluorine using low-temperature electrolysis, a process still employed for modern production. Industrial synthesis of fluorine gas for uranium enrichment, its largest application, began during the Manhattan Project in World War II. This video is targeted to blind users. Attribution: Article text available under CC-BY-SA Creative Commons image source in video
Views: 1155 Audiopedia
Endocrine System
 
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This video is meant to act as an index for topics from the FIRST AID for the USMLE STEP 1 (2016). If you find any errors or broken links, leave me a comment! Good luck in school!! Topic list: Embryology - Thyroid Development Anatomy - Adrenal Cortex and Medulla - Pituitary Gland Physiology - Insulin - Glucagon - Hypothalamic-Pituitary Hormones - Prolactin - Growth Hormone -Somatotropin - Appetitie Regulation - Antidiuretic Hormone - Adrenal Steroids and Congenital Adrenal Hyperplasias - Cortisol - Calcium Homeostasis - Vitamin D -Cholecalciferol - Parathyroid Hormone - Calcitonin - Thyroid Hormones -T3, T4 - Signaling Pathways of Endocrine Hormones - Signaling Pathways of Steroid Hormones Pathology - Cushing Syndrome - Adrenal Insufficiency - Primary Adrenal Insufficiency - Hyperaldosteronism - Neuroendocrine Tumors - Neuroblastoma - Pheochromocytoma - Hypothyroidism vs. Hyperthyroidism - Causes of Goiter - Hypothyroidism - Hyperthyroidism - Thyroid Adenoma - Thyroid Cancer - Diagnosis of Parathyroid Disease - Hypoparathyroidism - Familial Hypocalciuric Hypercalcemia - Hyperparathyroidism - Pituitary Adenoma - Nelson Syndrome - Acromegaly - Laron Syndrome -Dwarfism - Diabetes Insipidus - Syndrome of Inappropriate Antidiuretic Hormone Secretion - Hypopituitarism - Diabetes Mellitus - Type 1 vs. Type 2 Diabetes Mellitus - Diabetic Ketoacidosis - Hyperosmolar Hyperglycemia Nonketotic Syndrome - Glucagonoma - Insulinoma - Somatostatinoma - Carcinoid Syndrome - Zollinger-Ellison Syndrome - Multiple Endocrine Neoplasias Pharmacology - Diabetes Mellitus Drugs - Thionamides - Levothyroxine T4, Triiodothyronine T3 - Hypothalamic and Pituitary Drugs - Demeclocycline - Glucocorticoids - Fludrocortisone - Cinacalcet Hey guys and gals! I'm constantly trying to make and find better videos for you to study from (it’s not easy)! You can help by suggesting any good videos you've seen in the comments below! Good luck in school!! -~-~~-~~~-~~-~- Please watch: "Understand the Arterial Blood Gas " https://www.youtube.com/watch?v=1TnykLis7nA -~-~~-~~~-~~-~-
Views: 1096 Med Immersion
25: Antenatal Steroids - Part 1
 
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In this episode Dr. Øystein Bergøy discusses the evidence behind antenatal corticosteroids for women at risk for preterm delivery.  feedback@obgyn.fm Uptodate articles on RDS 1 2 3   Liggins 1972 paper 2013 cochrane analysis on type of steroid and regimens 2017 cochrane analysis of antenatal corticosteroids for accelerating fetal lung maturation    2015 cochrane analysis of repeat antenatal corticosteroids 2003 meta analysis of the effects of a single course corticosteroid compared to placebo/no treatment were delivery was 7 days or more after the treatment was given website of the EPICE cohort Study from EPICE looking at effects of steroids and time to delivery  ACOG guidelines on fetal maturation  NICE guidelines on risk of premature labour Norwegian guidelines on risk of premature labour
Views: 70 The Ob/Gyn Podcast
Dr.Ahmed Abd El-Rahman - Hormones 4 - TopicCorticosteroids - Part 3
 
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Subject : Pharmacology Tuesday - 18th, March 2014 Contents :- * Corticosteroids - Aldosterone - Des-Oxy-Corticosterone - Fludrocortisone Acetate *ACTH -Hydrocortisone *Theraputic uses of Glucocorticoids
Addison's disease
 
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Addison's disease is a rare, chronic endocrine disorder in which the adrenal glands do not produce sufficient steroid hormones . It is characterised by a number of relatively nonspecific symptoms, such as abdominal pain and weakness, but under certain circumstances, these may progress to Addisonian crisis, a severe illness which may include very low blood pressure and coma. This video targeted to blind users. Attribution: Article text available under CC-BY-SA Creative Commons image source in video
Views: 2413 encyclopediacc
Dr.Ahmed Abd El-Rahman - Hormones 4 - TopicCorticosteroids - Part 2
 
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Subject : Pharmacology Tuesday - 18th, March 2014 Contents :- * Corticosteroids - Aldosterone - Des-Oxy-Corticosterone - Fludrocortisone Acetate *ACTH -Hydrocortisone *Theraputic uses of Glucocorticoids
Hormonal Imbalance | On Call with the Prairie Doc | April 12, 2018
 
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Dr. Rick Holm, The Prairie Doc, is joined by Mark Oppenheimer, M.D., to discuss Hormonal Imbalance. They touch on the normal hormone functions of thyroid, pancreas, adrenal gland, pituitary, when there is too much or too little hormone. The will also talk about what treatments are available to slow down hormone excess, or replace hormone depletion. Mark J. Oppenheimer, M.D. has been practicing endocrinology in Sioux Falls since 1989. He opened Oppenheimer Endocrinology in 2005. Since then, the practice has grown to serve patients across the Midwest. Dr. Oppenheimer is also a clinical associate professor at the Sanford School of Medicine of the University of South Dakota. Learn more: https://www.PrairieDoc.org Like us on Facebook: https://www.facebook.com/ThePrairieDoc Follow us on Twitter: https://www.twitter.com/ThePrairieDoc Follow us on Instagram: https://www.instagram.com/ThePrairieDoc
Views: 52 The Prairie Doc